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Neurogenic Obesity After Spinal Cord Injury
Neurogenic Obesity after Spinal Cord Injury
Neurogenic Obesity after Spinal Cord Injury
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Video Summary
In this video, the presenters discuss the issue of neurogenic obesity after spinal cord injury (SCI). They define neurogenic obesity as a type of obesity that is unique to individuals with SCI and is characterized by a high percentage of body fat rather than traditional measures like BMI or waist circumference. They explain that several factors contribute to the development of neurogenic obesity, including motor paralysis below the level of injury, obligatory sarcopenia (loss of metabolically active tissue below the level of injury), sympathetic dysfunction, anabolic insufficiency, and blunted satiety. The presenters also discuss the pathophysiology of obesity in general and its relationship to metabolic syndrome. They explain that adipose tissue, which was once thought of as merely a storage organ for fat, is now recognized as an active endocrine organ that secretes hormones and plays a role in homeostasis. They also discuss the relationship between obesity and metabolic syndrome, which includes insulin resistance, hypertension, dyslipidemia, and other comorbidities. The presenters then explain the concept of energy balance and how it is affected in individuals with SCI. They highlight the reduced basal metabolic rate and resting metabolic rate in this population, as well as the lower thermic effect of both food and physical activity. They emphasize that individuals with SCI often consume more calories than they expend, leading to a positive energy balance and weight gain. The presenters suggest dietary interventions to manage obesity in individuals with SCI, such as following a low-energy, nutrient-dense diet that focuses on fruits, vegetables, whole grains, lean proteins, and low-fat dairy. They also discuss the importance of exercise in managing obesity and recommend aerobic and resistance training modalities. The presenters note that exercise may have limited impact on energy expenditure in individuals with SCI, but can still contribute to overall health and fitness. They mention the importance of interventions targeting both energy intake and expenditure, as well as the need for further research on specific exercise and dietary recommendations for individuals with SCI. Overall, the presenters provide a comprehensive overview of neurogenic obesity after SCI and highlight the challenges and potential strategies for managing this condition.
Keywords
neurogenic obesity
spinal cord injury
body fat
sarcopenia
metabolic syndrome
adipose tissue
energy balance
weight gain
dietary interventions
exercise
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